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Clomid or tamoxifen for a pct have you ever researched with pro hormones or gear or sarms and you’ve heard the term tct and maybe because it’s the interesting part you’ve been paying closer attention to the compounds right whether it be austrian or lgd in the world or rad or some other sign or in the gear side of things where you’re talking about 19 doors or
You’re talking about dht derivative drugs and talking about testosterone specifically there’s a lot of cool out there but not much focus is put on the pct but the two that tend to always come up for pct is clomid or clomiphene and tamoxifen which one is better we’re going to talk about that in this video but first if this is your first time here what’s good
My name is sam for those of you who are returning it’s good to freaking see you anybody who wants to support the channel jump down below grab some triggered uh products and um let’s get into it so you’re gonna probably learn something you may not be aware of it’ll probably get you to always choose a different direction than you may have in the past before so let’s
Start with tamoxifen and not all the data that i’m going to talk about is from these this is wikipedia super easy to pull out for these videos right so uh tamoxifen that’s a song that was developed back in the 1960s tamoxifen is used mainly on women but in men what it does is it binds to the estrogen receptor in your chest because it was used for women in breast
Sensitive cancer so like my mom had breast cancer right so the doctor gave her tamoxifen after her surgery because it helps slow down the growth or stop the growth of estrogen-sensitive cancer like breast cancer there’s certain ones that are not estrogen-sensitive so they came up with this and it binds to those estrogen receptors in the chest and it works as
An antagonist that means it just binds to the receptor so that nothing else can bind to it versus an agonist will bind to it so that it activates that result so it binds these receptors and it stops estrogen from getting to them so the body has a response to that in return in relation to the loop that kickstart so there’s a hpta access right that is a positive
Stimulus to the hypothalamus the pituitary the testicular axis that’s the function that drives your ionotropic to your fsh and lh they’re going on your balls your balls producing testosterone and this positively stimulates that right because it’s it’s trying to give our body feedback by having the estrogen on the receptor right because that’s part of the feedback
Process and it can’t get to that receptor because tamoxifen is there it’s like get out of your you get on your bench right so it tells the body that we need more of it and it starts producing more that’s what we want when we look at a pct so let’s look at clomiphene right well clothing you’re going to see a lot more data out there you probably see more doctors
Utilizing clomiphene because there’s more data on clomiphene um it was i don’t know when this one was actually started but it was approved here medically in 1967 so it’s clearly found probably at least a decade before that before tamoxifen and there’s more data in terms of tests on men with clomiphene i believe than the rest tamoxifen or there’s just a stigma
Or like a common nomenclature that people have been used for so long that people somehow think that clomid is like a good thing when it’s not so it’s in the same category as a serm right it’s a selective estrogen receptor modulator it’s going to go to the receptor and then what does it do with the receptor well clothing is made up of two parts and those two
Parts they go to the receptors those estrogen receptors remember tamoxifen is also a cern and it goes to the receptor and it sits on the receptor so that nothing else can get on there like estrogen right well clothing has two parts and so part of clovifi this is the 60 ish uh clomiphene that is this is made up of something called enclomophy remember how it
Said that one was that tamoxifen was a antagonist instead of an agonist meaning that tamoxifen binds to the receptor and it blocks it right it’s blocking this antagonism this has the same effect it binds to the receptor and it blocks estrogen from getting there oh that’s perfect well let’s just hope the second part of comed also does that because that’s the
Process that kicks our mind into go or our body responds to make more testosterone right which is then going to lead us to more estrogen which is why the body’s responding in that way well the other 30 something percent of clomid is something called end clomiphene i’m sorry uh zuclomaphine the first part is enclosing that’s 60 something percent and then this
Is oh here we go here’s a mixture of it 38 of it is zoochlomathine and 62 percent is enclosing right so that they have most of the good stuff right 62 is good stuff well what is this is this also good well this my friends is exactly what we don’t want zuma also selectively binds to the energy the estrogen receptor but it’s an agonist what does that mean so it
Binds to the receptor and it activates it like estrogen so what we’re effectively telling our body from that strategy is the opposite or the suppression of the hpt access remember how there’s a positive stimulus with tamoxifen right it’s stimulating your body to produce more testosterone well it’s the exact opposite with zoochomaphate meaning it pretends like
It’s estrogen in your body you don’t want that for pct my friends not a good idea that is not a good idea that’s one of the reasons why there’s so many reports of people dudes with side effects with clomiphene now you know why so just to wrap it up tamoxifen or clothing every day to my skin straight up 100 percent way better than climate and if you found this
Video helpful if you learned something in this video take a second share with somebody else there’s not a lot of people talking about this and call mafina’s other trash so take a second educate them help the channel out appreciate your peace
Transcribed from video
Clomid vs Tamoxifen – which best for PCT? By Sam Stolt
